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Cardiology · Spontaneous Coronary Artery Dissection

The Anatomy of SCAD: Tears, Bruises, and Angiograms

At a Glance

SCAD is a sudden tear or blood collection within a coronary artery wall that can restrict blood flow. Angiograms classify its pattern, while OCT or IVUS may clarify uncertain cases; the cath report records the vessel, type, flow, and management.

Understanding what happened inside your heart requires looking at the architecture of your arteries. While most heart attacks are caused by a slow buildup of plaque (fat and cholesterol), Spontaneous Coronary Artery Dissection (SCAD) is a sudden, structural failure of the artery wall itself [1][2].

The Biological Mechanism: Bruise vs. Plaque

In a traditional heart attack, a “pimple” of plaque ruptures, causing a blood clot to form and block the vessel. SCAD is entirely different. It starts inside the layers of the artery wall [2].

Think of your artery like a three-layered garden hose. In SCAD, blood forced into the middle layer creates a intramural hematoma (a localized bruise or collection of blood) [3]. This bruise is thought to happen in two ways, though mechanistic models cannot always prove which occurred in a specific patient:

  1. Inside-Out: A tiny tear (an intimal tear) develops in the inner lining, allowing blood to push into the wall and peel the layers apart [4].
  2. Outside-In: A tiny vessel within the wall itself (the vasa vasorum) bleeds, creating a bruise that grows and squashes the “true” channel where blood is supposed to flow [5][3].

Regardless of how it starts, the result is a “false” channel that narrows or blocks the “true” channel, starving the heart muscle of oxygen [1].

Why It Happens: Associations and Risks

SCAD is multifactorial, and an identifiable arteriopathy is not found in everyone. However, several important factors are associated with or may contribute to the condition [6].

  • Fibromuscular Dysplasia (FMD): This is the most common association. FMD is a nonatherosclerotic, noninflammatory arterial disease involving abnormal arterial structure, making arteries more likely to dissect [7][8]. Because FMD can affect other arteries, doctors usually recommend evaluating your major vessels (from head to pelvis) using a CTA or MRA scan [9][10].
  • Pregnancy and Hormones: Pregnancy-associated SCAD is a recognized context, most common in the late stages of pregnancy or the first month postpartum [11]. Hormonal and hemodynamic variations have been proposed as contributors [12].
  • Stress and Genetics: Extreme emotional or physical exertion are frequently reported preceding events, though this does not mean you caused your SCAD. In a small number of cases, a genetic disorder affecting connective tissue may be found [13][14].

The Angiographic Types

When you have a coronary angiogram (a procedure where dye is injected into your heart arteries), doctors look for specific patterns to classify the SCAD. The widely used Saw classification system includes Types 1, 2, and 3 (with Type 2 sometimes subdivided), and occasionally Type 4 is used to denote a complete occlusion [15][16].

  • Type 1 (The Classic Tear): The dye clearly enters a tear, showing multiple “channels” or visible contrast staining in the artery lining [17].
  • Type 2 (The Long Narrowing): The most common type. The artery looks long and diffusely narrowed because a long intramural hematoma is compressing it from the inside [15].
  • Type 3 (The Plaque Mimic): This looks like a focal tubular stenosis that could easily be mistaken for traditional plaque, making it the hardest to diagnose [15].
  • Type 4 (Complete Occlusion): The artery is completely shut off [16].

Note: Angiographic type terminology varies by region and does not by itself determine your prognosis or dictate exactly how your artery will heal. Your treating cardiologist interprets the entire angiogram.

Advanced Imaging: OCT and IVUS

If your angiogram is unclear (common in Types 2 and 3), doctors may use “inside-the-vessel” imaging [17]:

  • OCT (Optical Coherence Tomography): Uses light waves to take high-resolution cross-sectional pictures. It can clearly show the hematoma and the tear [18].
  • IVUS (Intravascular Ultrasound): Uses sound waves to see through the artery wall.

The Risk: While these tools are powerful, they are used cautiously. Because the artery wall is already fragile, inserting the imaging probe or injecting the dye required can sometimes extend the dissection or cause a new tear [19][20]. The clinical team weighs the diagnostic benefit against these procedural risks.

Your Cath Lab Report Checklist

Your “Cath Report” is the official record of your diagnosis. A complete report should ideally document the following terms. You should request the actual angiographic images (on a CD or drive) as well as the written report.

  • [ ] Vessel(s) Involved: Which artery was affected? (e.g., LAD for Left Anterior Descending, RCA for Right Coronary Artery) [21]
  • [ ] Angiographic Type: Was it Type 1, 2, 3, or 4? [17]
  • [ ] Lesion Location: Was the tear at the beginning (proximal) or end (distal) of the vessel? [22]
  • [ ] Blood Flow (TIMI Grade): How well was blood moving past the injury? (TIMI 3 indicates normal blood flow) [17]
  • [ ] Imaging Used: If OCT or IVUS was used, what did it show? [23]
  • [ ] Management Choice: Why did the team choose “conservative management” vs. a stent or bypass? [24]

Common questions in this guide

What is SCAD, and how is it different from a typical heart attack?
SCAD happens when a tear or bleeding within the wall of a coronary artery creates a bruise that narrows or blocks the channel carrying blood. A typical heart attack is usually caused by plaque buildup that ruptures and triggers a clot, whereas SCAD is a structural problem in the artery wall.
What does an intramural hematoma mean in SCAD?
An intramural hematoma is a collection of blood inside the layers of the artery wall. It can expand and compress the normal channel, reducing oxygen-rich blood reaching the heart muscle. It may form after a small inner-wall tear or bleeding from tiny vessels within the artery wall.
What do the different SCAD angiogram types show?
Type 1 shows a visible tear or more than one channel, Type 2 shows a long area of narrowing, and Type 3 is a short narrowing that can resemble plaque. Type 4 describes a complete blockage and is used in some classification systems; the type alone does not determine how the artery will heal or what treatment is needed.
Why might doctors use OCT or IVUS during a SCAD evaluation?
OCT uses light and IVUS uses sound waves to create images from inside the artery. They can help show a wall bruise or tear when an angiogram is unclear. Because SCAD arteries can be fragile, placing a probe or injecting dye can extend the dissection or cause a new tear, so the clinical team weighs the benefits against these risks.
Why might someone with SCAD be checked for fibromuscular dysplasia?
Fibromuscular dysplasia, or FMD, is the most common condition associated with SCAD and can affect arteries outside the heart. Clinicians often consider imaging arteries from the head to the pelvis with CTA or MRA, although the appropriate screening plan depends on the person's medical history.
What information should be included in a SCAD cath report?
The report should identify the affected artery, SCAD angiographic type, lesion location, blood flow grade, and whether OCT or IVUS was used. It should also document why conservative management, a stent, or bypass surgery was selected; keeping the written report and angiographic images can help future clinicians.
Can pregnancy or stress be linked to SCAD?
Pregnancy-associated SCAD is a recognized context, particularly during the later stages of pregnancy or the first month after delivery. Extreme physical or emotional exertion is often reported before SCAD, but this does not prove it caused the event and does not mean the patient is to blame.

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References

References (24)
  1. 1

    Spontaneous Coronary Artery Dissection: Advances in Understanding and Management in Women.

    Chai J, Saw J

    Current atherosclerosis reports 2026; (28(1)).

    PMID: 42096100
  2. 2

    Chest Pain: Wellens Syndrome Due to Spontaneous Dissection of the Left Anterior Descending Coronary Artery - A Case Report and Literature Review.

    Clemente G, Quaranta C, Basso MG, et al.

    Reviews in cardiovascular medicine 2024; (25(2)):70 doi:10.31083/j.rcm2502070.

    PMID: 39077363
  3. 3

    On the provenance of spontaneous coronary artery dissection.

    Graham RM, Hayes SN, McGrath-Cadell L, et al.

    European heart journal open 2026; (6(2)):oeag066 doi:10.1093/ehjopen/oeag066.

    PMID: 42027788
  4. 4

    Spontaneous coronary artery dissection: review, case report and analysis of COVID-19-related cases.

    Cosma J, Russo A, Ferradini V, et al.

    Minerva cardiology and angiology 2024; (72(3)):251-265 doi:10.23736/S2724-5683.22.06195-6.

    PMID: 36847436
  5. 5

    Early Natural History of Spontaneous Coronary Artery Dissection.

    Waterbury TM, Tweet MS, Hayes SN, et al.

    Circulation. Cardiovascular interventions 2018; (11(9)):e006772 doi:10.1161/CIRCINTERVENTIONS.118.006772.

    PMID: 30354594
  6. 6

    Pathology of Genetic Variants in Spontaneous Coronary Artery Dissection.

    Tanaka T, Kawakami R, Gaynor BJ, et al.

    Circulation 2026; (154(14)):1309-1321 doi:10.1161/CIRCULATIONAHA.126.080495.

    PMID: 42677491
  7. 7

    Extracoronary Arterial Pathologies in Patients With Spontaneous Coronary Artery Dissection.

    Bcharah H, Bcharah G, Nabi HA, et al.

    The American journal of cardiology 2026; (258()):151-157 doi:10.1016/j.amjcard.2025.08.042.

    PMID: 40912385
  8. 8

    Canadian Spontaneous Coronary Artery Dissection Cohort Study: 3-Year Outcomes.

    Saw J, Starovoytov A, Aymong E, et al.

    Journal of the American College of Cardiology 2022; (80(17)):1585-1597 doi:10.1016/j.jacc.2022.08.759.

    PMID: 36265953
  9. 9

    Fibromuscular Dysplasia: A Focused Review for the Cardiologist.

    Petropoulos T, Shah A, Dueck A, et al.

    CJC open 2024; (6(11)):1274-1288 doi:10.1016/j.cjco.2024.07.014.

    PMID: 39582709
  10. 10

    Spontaneous Coronary Artery Dissection and Fibromuscular Dysplasia: A Case Series and Genetic Links.

    Le Q, Bhandari A, Fleischer J, Murthy A

    Cureus 2024; (16(2)):e54105 doi:10.7759/cureus.54105.

    PMID: 38487126
  11. 11

    Spontaneous Coronary Artery Dissection Associated With Pregnancy.

    Tweet MS, Hayes SN, Codsi E, et al.

    Journal of the American College of Cardiology 2017; (70(4)):426-435 doi:10.1016/j.jacc.2017.05.055.

    PMID: 28728686
  12. 12

    Pregnancy-Associated Spontaneous Coronary Artery Dissection: A Report of the iSCAD Registry.

    Koczo A, Grodzinsky A, Kim ESH, et al.

    JAMA cardiology 2026; (11(6)):534-543 doi:10.1001/jamacardio.2026.1009.

    PMID: 41904991
  13. 13

    Canadian spontaneous coronary artery dissection cohort study: in-hospital and 30-day outcomes.

    Saw J, Starovoytov A, Humphries K, et al.

    European heart journal 2019; (40(15)):1188-1197 doi:10.1093/eurheartj/ehz007.

    PMID: 30698711
  14. 14

    Prospective Cardiovascular Genetics Evaluation in Spontaneous Coronary Artery Dissection.

    Kaadan MI, MacDonald C, Ponzini F, et al.

    Circulation. Genomic and precision medicine 2018; (11(4)):e001933 doi:10.1161/CIRCGENETICS.117.001933.

    PMID: 29650765
  15. 15

    Spontaneous Coronary Artery Dissection: Diagnosis and Management.

    Yeo I, Feldman DN, Kim LK

    Current treatment options in cardiovascular medicine 2018; (20(3)):27 doi:10.1007/s11936-018-0622-2.

    PMID: 29549452
  16. 16

    Clinical and angiographic features of SCAD type 4.

    Mori R, Macaya F, Giacobbe F, et al.

    International journal of cardiology 2023; (377()):22-25 doi:10.1016/j.ijcard.2023.01.067.

    PMID: 36716971
  17. 17

    Angiographic appearance of spontaneous coronary artery dissection with intramural hematoma proven on intracoronary imaging.

    Saw J, Mancini GB, Humphries K, et al.

    Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions 2016; (87(2)):E54-61 doi:10.1002/ccd.26022.

    PMID: 26198289
  18. 18

    Optical Coherence Tomography and Coronary Dissection: Precious Tool or Useless Surplus?

    Barbieri L, D'Errico A, Avallone C, et al.

    Frontiers in cardiovascular medicine 2022; (9()):822998 doi:10.3389/fcvm.2022.822998.

    PMID: 35433885
  19. 19

    Optical Coherence Tomography for Spontaneous Coronary Artery Dissection Assessment: The Flush-Free OCT Technique.

    Al-Abcha A, Simard T, Tweet MS, et al.

    JACC. Case reports 2025; (30(21)):104398 doi:10.1016/j.jaccas.2025.104398.

    PMID: 40750136
  20. 20

    A case of spontaneous coronary artery dissection complicated with pseudoaneurysm healed by medical treatment: Follow-up by multidetector computed tomography.

    Onoda N, Izumi K

    Journal of cardiology cases 2018; (18(5)):156-159 doi:10.1016/j.jccase.2018.06.005.

    PMID: 30416614
  21. 21

    Spontaneous coronary artery dissection in Spain: clinical and angiographic characteristics, management, and in-hospital events.

    García-Guimaraes M, Bastante T, Macaya F, et al.

    Revista espanola de cardiologia (English ed.) 2021; (74(1)):15-23 doi:10.1016/j.rec.2020.04.002.

    PMID: 32418854
  22. 22

    Characteristics and outcomes of spontaneous coronary artery dissection of the left anterior descending artery.

    Marschall A, García-Guimarães M, Sanz-Ruiz R, et al.

    EuroIntervention : journal of EuroPCR in collaboration with the Working Group on Interventional Cardiology of the European Society of Cardiology 2026; (22(16)):874-882 doi:10.4244/EIJ-D-25-01394.

    PMID: 42610336
  23. 23

    STEMI caused by spontaneous coronary artery dissection: prevalence, patient characteristics and outcomes.

    Malleier S, Gaede L, Jung S, et al.

    Clinical research in cardiology : official journal of the German Cardiac Society 2026; (115(10)):1845-1855 doi:10.1007/s00392-026-02900-y.

    PMID: 41885960
  24. 24

    Spontaneous coronary artery dissection: update 2019.

    Gilhofer TS, Saw J

    Current opinion in cardiology 2019; (34(6)):594-602 doi:10.1097/HCO.0000000000000671.

    PMID: 31567442

This page is for informational purposes only and does not constitute medical advice. Your cardiologist should interpret your SCAD angiogram, imaging, and cath report in the context of your individual care.